Editing Accessory fruit (section)

== Research ==
Current research has proposed that a single class of [[gene]]s may be responsible for regulating accessory fruit formation and ripening.<ref>{{cite journal |last1=Ireland |first1=Hilary |last2=Yao |first2=Jia-Long |last3=Tomes |first3=Sumathi |last4=Sutherland |first4=Paul |last5=Nieuwenhuizen |first5=Niels |last6=Gunaseelan |first6=Kularajathevan |last7=Winz |first7=Robert |last8=David |first8=Karine |last9=Schaffer |first9=Robert |title=Apple SEPALLATA1/2-like genes control fruit flesh development and ripening |journal=The Plant Journal |date=13 December 2012 |volume=73 |issue=6 |pages=1004–1056 |doi=10.1111/tpj.12094 |pmid=23236986 |doi-access= }}</ref> A study using strawberries concluded that hormone signaling pathways involving [[gibberellic acid]] and [[auxin]] affect gene expression, and contribute to the initiation of accessory fruit development.<ref name=":1">{{cite journal |last1=Zhou |first1=Junhui |last2=Sittmann |first2=John |last3=Guo |first3=Lei |last4=Xiao |first4=Yuwei |last5=Huang |first5=Xiaolong |last6=Pulapaka |first6=Anuhya |last7=Liu |first7=Zhongchi |title=Gibberellin and auxin signaling genes RGA1 and ARF8 repress accessory fruit initiation in diploid strawberry |journal=Plant Physiology |date=March 2021 |volume=185 |issue=3 |pages=1059–1075 | pmid=33793929 | doi=10.1093/plphys/kiaa087 |pmc=8133647}}</ref> Metabolic modifications in different developing accessory fruit tissues are due to the varied distributions of compounds such as [[triterpenoids]] and [[steroid]]s.<ref>{{cite journal |last1=Dashbaldan |first1=Soyol |last2=Rogowska |first2=Agata |last3=Pączkowski |first3=Cezary |last4=Szakiel |first4=Anna |title=Distribution of Triterpenoids and Steroids in Developing Rugosa Rose (Rosarugosa Thunb.) Accessory Fruit |journal=Molecules |date=25 August 2021 |volume=26 |issue=17 |page=5158 | pmid=34500591| doi=10.3390/molecules26175158 | pmc=8433923 |doi-access=free }}</ref>