Editing Urinary system (section)

====Regulation of concentration and volume====

The urinary system is under influence of the [[circulatory system]], [[nervous system]], and [[endocrine system]].

[[Aldosterone]] plays a central role in regulating blood pressure through its effects on the kidney. It acts on the distal tubules and collecting ducts of the nephron and increases reabsorption of sodium from the glomerular filtrate. Reabsorption of sodium results in retention of water, which increases blood pressure and blood volume. Antidiuretic hormone (ADH), is a [[neurohypophysial hormone]] found in most [[mammals]]. Its two primary functions are to retain water in the body and [[vasoconstriction]]. Vasopressin regulates the body's [[Water retention (medicine)|retention of water]] by increasing water reabsorption in the collecting ducts of the kidney nephron.<ref name="isbn0-387-30348-0">{{cite book |vauthors=Caldwell HK, ((Young WS III)), Lajtha A, Lim R | title = Handbook of Neurochemistry and Molecular Neurobiology: Neuroactive Proteins and Peptides | edition = 3rd | publisher = Springer | location = Berlin | year = 2006 | chapter =  Oxytocin and Vasopressin: Genetics and Behavioral Implications | chapter-url= http://refworks.springer.com/mrw/fileadmin/pdf/Neurochemistry/0387303480C25.PDF | pages = 573–607| isbn = 0-387-30348-0 }}</ref> Vasopressin increases water permeability of the kidney's collecting duct and distal convoluted tubule by inducing translocation of aquaporin-CD water channels in the kidney nephron collecting duct plasma membrane.<ref name="pmid7532304">{{cite journal |vauthors=Nielsen S, Chou CL, Marples D, Christensen EI, Kishore BK, Knepper MA | title = Vasopressin increases water permeability of kidney collecting duct by inducing translocation of aquaporin-CD water channels to plasma membrane | journal = Proc. Natl. Acad. Sci. U.S.A. | volume = 92 | issue = 4 | pages = 1013–7 |date=February 1995 | pmid = 7532304 | pmc = 42627 | doi = 10.1073/pnas.92.4.1013| bibcode = 1995PNAS...92.1013N | doi-access = free }}</ref>