Editing Cardiomyopathy (section)

==Mechanism==
The pathophysiology of cardiomyopathies is better understood at the cellular level with advances in molecular techniques. Mutant proteins can disturb cardiac function in the contractile apparatus (or mechanosensitive complexes). Cardiomyocyte alterations and their persistent responses at the cellular level cause changes that are correlated with sudden cardiac death and other cardiac problems.<ref>{{Cite journal |last=Harvey |first=Pamela A. |last2=Leinwand |first2=Leslie A. |date=2011-08-08 |title=Cellular mechanisms of cardiomyopathy |journal=The Journal of Cell Biology |volume=194 |issue=3 |pages=355–365 |doi=10.1083/jcb.201101100 |issn=0021-9525 |pmc=3153638 |pmid=21825071}}</ref>

Cardiomyopathies are generally varied individually. Different factors can cause cardiomyopathies in adults as well as children. For example, dilated cardiomyopathy in adults is associated with ischemic cardiomyopathy, hypertension, valvular diseases, and genetics. In children, neuromuscular diseases such as Becker muscular dystrophy or X-linked genetic disorder, are directly linked with cardiomyopathies.<ref>{{Cite journal |last=Braunwald |first=Eugene |date=2017-09-15 |title=Cardiomyopathies: An Overview |journal=Circulation Research |volume=121 |issue=7 |pages=711–721 |doi=10.1161/CIRCRESAHA.117.311812 |issn=1524-4571 |pmid=28912178 |s2cid=36384619 |doi-access=free}}</ref>