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===Antipsychotics=== Many [[antipsychotic]]s bind to and modulate [[serotonin receptor]]s, including the serotonin [[5-HT1A receptor|5-HT<sub>1A</sub>]], [[5-HT2A receptor|5-HT<sub>2A</sub>]], [[5-HT2B receptor|5-HT<sub>2B</sub>]], [[5-HT2C receptor|5-HT<sub>2C</sub>]], [[5-HT6 receptor|5-HT<sub>6</sub>]], and [[5-HT7 receptor|5-HT<sub>7</sub> receptor]]s, among others.<ref name="Meltzer1999">{{cite journal | vauthors = Meltzer HY | title = The role of serotonin in antipsychotic drug action | journal = Neuropsychopharmacology | volume = 21 | issue = 2 Suppl | pages = 106Sβ115S | date = August 1999 | pmid = 10432496 | doi = 10.1016/S0893-133X(99)00046-9 | url = }}</ref><ref name="Meltzer2012">{{cite book | vauthors = Meltzer HY | chapter = Serotonergic Mechanisms as Targets for Existing and Novel Antipsychotics | title = Handb Exp Pharmacol | series = Handbook of Experimental Pharmacology | volume = 212| issue = 212 | pages = 87β124 | date = 2012 | pmid = 23129329 | doi = 10.1007/978-3-642-25761-2_4 | isbn = 978-3-642-25760-5 | chapter-url = }}</ref> Activation of serotonin 5-HT<sub>1A</sub> receptors and blockade of serotonin 5-HT<sub>2A</sub> receptors may contribute to the therapeutic antipsychotic effects of these agents, whereas antagonism of serotonin 5-HT<sub>2C</sub> receptors has been especially implicated in [[side effect]]s of antipsychotics.<ref name="Meltzer1999" /><ref name="Meltzer2012" />
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