Editing Cerebellum (section)

===Pain===
There is a general consensus that the cerebellum is involved in pain processing.<ref>{{cite journal | vauthors = Moulton EA, Schmahmann JD, Becerra L, Borsook D | title = The cerebellum and pain: passive integrator or active participator? | journal = Brain Research Reviews | volume = 65 | issue = 1 | pages = 14–27 | date = October 2010 | pmid = 20553761 | pmc = 2943015 | doi = 10.1016/j.brainresrev.2010.05.005 }}</ref><ref>{{cite journal | vauthors = Baumann O, Borra RJ, Bower JM, Cullen KE, Habas C, Ivry RB, Leggio M, Mattingley JB, Molinari M, Moulton EA, Paulin MG, Pavlova MA, Schmahmann JD, Sokolov AA | title = Consensus paper: the role of the cerebellum in perceptual processes | journal = Cerebellum | volume = 14 | issue = 2 | pages = 197–220 | date = April 2015 | pmid = 25479821 | pmc = 4346664 | doi = 10.1007/s12311-014-0627-7 }}</ref> The cerebellum receives pain input from both descending cortico-cerebellar pathways and ascending spino-cerebellar pathways, through the pontine nuclei and inferior olives. Some of this information is transferred to the motor system inducing a conscious motor avoidance of pain, graded according to pain intensity.

These direct pain inputs, as well as indirect inputs, are thought to induce long-term pain avoidance behavior that results in chronic posture changes and consequently, in functional and anatomical remodeling of vestibular and proprioceptive nuclei. As a result, chronic neuropathic pain can induce macroscopic anatomical remodeling of the hindbrain, including the cerebellum.<ref name="ReferenceA"/> The magnitude of this remodeling and the induction of neuron progenitor markers suggest the contribution of [[Adult neurogenesis#Tracking neurogenesis|adult neurogenesis]] to these changes.