Editing Human pathogen (section)

===Viral===
{{Further|Viral disease}}
Pathogenic viruses are mainly those of the families of: ''[[Adenoviridae]],  [[Picornaviridae]], [[Herpesviridae]], [[Hepadnaviridae]], [[Coronaviridae]], [[Flaviviridae]], [[Retroviridae]], [[Orthomyxoviridae]], [[Paramyxoviridae]], [[Papovaviridae]], [[Polyomavirus]], [[Poxviridae]], [[Rhabdoviridae]]'', and ''[[Togaviridae]]''. Some notable pathogenic viruses cause [[smallpox]], [[influenza]], [[mumps]], [[measles]], [[chickenpox]], [[ebola]], and rubella. Viruses typically range between 20 and 300 nanometers in length.
<ref>{{Cite web |url=https://www.cdc.gov/ncidod/dvrd/spb/mnpages/dispages/ebola/qa.htm |title=Questions and Answers about Ebola Hemorrhagic Fever |access-date=2017-09-09 |archive-url=https://web.archive.org/web/20090506104535/http://www.cdc.gov/ncidod/dvrd/spb/mnpages/dispages/ebola/qa.htm |archive-date=2009-05-06 |url-status=dead | work = U.S. Centers for Disease Control and Prevention }}</ref>

This type of pathogen is not cellular, and is instead composed of either RNA ([[RNA|Ribonucleic acid]]) or DNA ([[DNA|Deoxyribonucleic acid]]) within a protein shell - the [[capsid]]. Pathogenic viruses infiltrate host cells and manipulate the organelles within the cell such as the [[Ribosome]]s, [[Golgi apparatus|Golgi Apparatus]], and [[Endoplasmic reticulum|Endoplasmic Reticulum]] in order to multiply which commonly results in the death of the host cell via cellular decay. All the viruses that were contained within the [[lipid bilayer]] of the cell membrane are then released into the [[Extracellular matrix|intercellular matrix]] to infect neighboring cells to continue the [[viral life cycle]].

[[White blood cell]]s surround and consume the virus using a mechanism known as [[phagocytosis]]<ref name="Nainu_2017">{{cite journal |last1= Nainu |first1= Firzan |last2= Shiratsuchi |first2= Akiko |last3= Nakanishi |first3= Yoshinobu| date=2017-09-28 |title=Induction of Apoptosis and Subsequent Phagocytosis of Virus-Infected Cells As an Antiviral Mechanism |journal= Frontiers in Immunology |volume=8 |issue=1220 |pages=954 |doi=10.3389/fimmu.2017.01220 |doi-access= free |pmid=29033939 |pmc= 5624992 }}</ref> (a type of [[endocytosis]])<ref name="Johnson_2002">{{cite book |last1=Alberts |first1=B. |last2=Johnson |first2=A. |last3=Lewis |first3=J.  |date=2002 |title=SMolecular Biology of the Cell. 4th edition. |url=https://www.ncbi.nlm.nih.gov/books/NBK26870/#:~:text=Phagocytosis%20is%20a%20special%20form,large%20endocytic%20vesicles%20called%20phagosomes. |location=New York, USA |publisher=Garland Science  |isbn=9780815332183}}</ref> within the [[extracellular matrix]] to reduce and fight the infection. The components within the [[white blood cell]] are responsible for destroying the virus and recycling its components for the body to use.<ref>{{Cite book |last=Melloy |first=Patricia G. |url=https://doi.org/10.1201/9781003172260 |title=Viruses and Society |date=2022-07-25 |publisher=CRC Press |isbn=978-1-003-17226-0 |location=Boca Raton}}</ref>