Editing Perception (section)

==== Time (chronoception) ====
{{main|time perception}}
[[Time perception|Chronoception]] refers to how the passage of [[time]] is perceived and experienced. Although the [[Time perception|sense of time]] is not associated with a specific [[sensory system]], the work of [[psychologist]]s and [[neuroscientist]]s indicates that human brains do have a system governing the perception of time,<ref name="Rao 2001">{{cite journal|vauthors=Rao SM, Mayer AR, Harrington DL|date=March 2001|title=The evolution of brain activation during temporal processing|journal=Nature Neuroscience|volume=4|issue=3|pages=317–23|doi=10.1038/85191|pmid=11224550|s2cid=3570715|url=https://escholarship.org/uc/item/80c4d02m }}</ref><ref>{{cite web|url=http://www.unisci.com/stories/20011/0227013.htm|title=Brain Areas Critical To Human Time Sense Identified|date=2001-02-27|publisher=UniSci – Daily University Science News}}</ref> composed of a highly distributed system involving the [[cerebral cortex]], [[cerebellum]], and [[basal ganglia]]. One particular component of the brain, the [[suprachiasmatic nucleus]], is responsible for the [[circadian rhythm]] (commonly known as one's "internal clock"), while other cell clusters appear to be capable of shorter-range timekeeping, known as an ''[[ultradian]] rhythm''.

One or more [[dopaminergic pathways]] in the [[central nervous system]] appear to have a strong modulatory influence on [[mental chronometry]], particularly [[Interval (time)|interval timing.]]<ref name="Amph-DA reaction time">{{cite journal|vauthors=Parker KL, Lamichhane D, Caetano MS, Narayanan NS|date=October 2013|title=Executive dysfunction in Parkinson's disease and timing deficits|journal=Frontiers in Integrative Neuroscience|volume=7|pages=75|doi=10.3389/fnint.2013.00075|pmc=3813949|pmid=24198770|quote=Manipulations of dopaminergic signaling profoundly influence interval timing, leading to the hypothesis that dopamine influences internal pacemaker, or "clock", activity. For instance, amphetamine, which increases concentrations of dopamine at the synaptic cleft advances the start of responding during interval timing, whereas antagonists of D2 type dopamine receptors typically slow timing;... Depletion of dopamine in healthy volunteers impairs timing, while amphetamine releases synaptic dopamine and speeds up timing.|doi-access=free}}</ref>