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===Anhydrobiosis=== Bdelloid rotifer females cannot produce resting eggs, but many can survive prolonged periods of adverse conditions after [[desiccation]]. This facility is termed [[anhydrobiosis]], and organisms with these capabilities are termed anhydrobionts. Under drought conditions, bdelloid rotifers contract into an inert form and lose almost all body water; when rehydrated they resume activity within a few hours. Bdelloids can survive the dry state for long periods, with the longest well-documented dormancy being nine years. Rotifers can also undergo other forms of cryptobiosis, notably cryobiosis which results from decreased temperatures. In 2021, researchers collected samples from remote Arctic locations containing rotifers which when thawed revealed living specimens around 24,000 years old.<ref>{{Cite journal |last1=Shmakova |first1=Lyubov |last2=Malavin |first2=Stas |last3=Iakovenko |first3=Nataliia |last4=Vishnivetskaya |first4=Tatiana |last5=Shain |first5=Daniel |last6=Plewka |first6=Michael |last7=Rivkina |first7=Elizaveta |date=June 2021 |title=A living bdelloid rotifer from 24,000-year-old Arctic permafrost |journal=Current Biology |volume=31 |issue=11 |pages=R712βR713 |doi=10.1016/j.cub.2021.04.077 |s2cid=235365588 |issn=0960-9822|doi-access=free |pmid=34102116 |bibcode=2021CBio...31.R712S }}</ref> While in other anhydrobionts, such as the [[brine shrimp]], this desiccation tolerance is thought to be linked to the production of [[trehalose]], a non-reducing disaccharide ([[sugar]]), bdelloids apparently cannot synthesise trehalose. In bdelloids, a major cause of the resistance to desiccation, as well as resistance to ionizing radiation, is a highly efficient mechanism for repairing the DNA double-strand breaks induced by these agents.<ref name="pmid25105197">{{cite journal |vauthors=Hespeels B, Knapen M, Hanot-Mambres D, Heuskin AC, Pineux F, LUCAS S, Koszul R, Van Doninck K |title=Gateway to genetic exchange? DNA double-strand breaks in the bdelloid rotifer Adineta vaga submitted to desiccation |journal=J. Evol. Biol. |volume=27 |issue=7 |pages=1334β45 |date=July 2014 |pmid=25105197 |doi= 10.1111/jeb.12326|url=https://hal-pasteur.archives-ouvertes.fr/pasteur-01420001/file/hespeels%20et%20al%202014.pdf|doi-access=free }}</ref> This repair mechanism likely involves mitotic recombination between homologous DNA regions.<ref name="pmid25105197" />
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