Editing Glaucoma (section)

===Secondary glaucoma===
[[Secondary glaucoma]] (H40.3-H40.6) encompasses numerous subtypes: inflammatory glaucoma (all types of uveitis, Fuchs heterochromic iridocyclitis); phacogenic glaucoma (angle-closure with mature [[cataract]], phacoanaphylactic glaucoma from [[lens capsule]] rupture, [[phacolytic glaucoma]], [[lens subluxation]]); glaucoma secondary to intraocular hemorrhage ([[hyphema]], [[Hemolysis|hemolytic]]/erythroclastic glaucoma); traumatic glaucoma (angle recession glaucoma); postsurgical glaucoma (aphakic pupillary block, ciliary block); neovascular glaucoma; drug-induced glaucoma (corticosteroid-induced, alpha-chymotrypsin); and glaucoma of miscellaneous origin (associated with [[Eye neoplasm|intraocular tumors]], [[Retinal detachment|retinal detachments]], severe chemical burns, [[essential iris atrophy]], or toxic glaucoma).

'''Neovascular glaucoma''', an uncommon type of glaucoma, is difficult or nearly impossible to treat, and is often caused by proliferative [[diabetic retinopathy]] (PDR) or [[central retinal vein occlusion]] (CRVO). It may also be triggered by other conditions that result in [[ischemia]] of the [[retina]] or [[ciliary body]]. Individuals with poor blood flow to the eye are highly at risk for this condition.

Neovascular glaucoma results when new, abnormal vessels begin developing in the angle of the eye that begin blocking the drainage. People with such condition begin to rapidly lose their eyesight. Sometimes, the disease appears very rapidly, especially after cataract surgery procedures.

'''Toxic glaucoma''' is open-angle glaucoma with an unexplained significant rise of intraocular pressure following unknown pathogenesis. Intraocular pressure can sometimes reach {{convert|80|mmHg|abbr=on}}. It characteristically manifests as ciliary body inflammation and massive trabecular edema that sometimes extends to Schlemm's canal. This condition is differentiated from malignant glaucoma by the presence of a deep and clear anterior chamber and a lack of aqueous misdirection. Also, the corneal appearance is not as hazy. A reduction in visual acuity can occur followed neuroretinal breakdown.