Editing Ataxia (section)

=== Non-hereditary cerebellar degeneration ===
Non-hereditary causes of cerebellar degeneration include chronic [[alcohol use disorder]], [[head injury]], [[paraneoplastic cerebellar degeneration|paraneoplastic]] and non-paraneoplastic [[autoimmunity|autoimmune]] ataxia,<ref name="Medusa head ataxia - Part 1">{{cite journal | vauthors = Jarius S, Wildemann B | title = 'Medusa-head ataxia': the expanding spectrum of Purkinje cell antibodies in autoimmune cerebellar ataxia. Part 1: Anti-mGluR1, anti-Homer-3, anti-Sj/ITPR1 and anti-CARP VIII | journal = Journal of Neuroinflammation | volume = 12 | issue = 1 | pages = 166 | date = September 2015 | pmid = 26377085 | pmc = 4574226 | doi = 10.1186/s12974-015-0356-y | doi-access = free }}</ref><ref name="Medusa head ataxia - Part 2">{{cite journal | vauthors = Jarius S, Wildemann B | title = 'Medusa head ataxia': the expanding spectrum of Purkinje cell antibodies in autoimmune cerebellar ataxia. Part 2: Anti-PKC-gamma, anti-GluR-delta2, anti-Ca/ARHGAP26 and anti-VGCC | journal = Journal of Neuroinflammation | volume = 12 | issue = 1 | pages = 167 | date = September 2015 | pmid = 26377184 | pmc = 4574118 | doi = 10.1186/s12974-015-0357-x | doi-access = free }}</ref><ref name="Medusa head ataxia - Part 3">{{cite journal | vauthors = Jarius S, Wildemann B | title = 'Medusa head ataxia': the expanding spectrum of Purkinje cell antibodies in autoimmune cerebellar ataxia. Part 3: Anti-Yo/CDR2, anti-Nb/AP3B2, PCA-2, anti-Tr/DNER, other antibodies, diagnostic pitfalls, summary and outlook | journal = Journal of Neuroinflammation | volume = 12 | issue = 1 | pages = 168 | date = September 2015 | pmid = 26377319 | pmc = 4573944 | doi = 10.1186/s12974-015-0358-9 | doi-access = free }}</ref> [[high-altitude cerebral edema]],<ref name="Sharma_2019">{{cite journal | vauthors = Sharma R, Cramer NP, Perry B, Adahman Z, Murphy EK, Xu X, Dardzinski BJ, Galdzicki Z, Perl DP, Dickstein DL, Iacono D | title = Chronic Exposure to High Altitude: Synaptic, Astroglial and Memory Changes | journal = Scientific Reports | volume = 9 | issue = 1 | pages = 16406 | date = November 2019 | pmid = 31712561 | pmc = 6848138 | doi = 10.1038/s41598-019-52563-1 | bibcode = 2019NatSR...916406S }}</ref> [[celiac disease]],<ref name="Hermaszewski_1991">{{cite journal | vauthors = Hermaszewski RA, Rigby S, Dalgleish AG | title = Coeliac disease presenting with cerebellar degeneration | journal = Postgraduate Medical Journal | volume = 67 | issue = 793 | pages = 1023–1024 | date = November 1991 | pmid = 1775412 | pmc = 2399130 | doi = 10.1136/pgmj.67.793.1023 }}</ref> [[normal-pressure hydrocephalus]],<ref name="Lv_2022">{{cite journal | vauthors = Lv M, Yang X, Zhou X, Chen J, Wei H, Du D, Lin H, Xia J | title = Gray matter volume of cerebellum associated with idiopathic normal pressure hydrocephalus: A cross-sectional analysis | journal = Frontiers in Neurology | volume = 13 | pages = 922199 | date = 2022-09-07 | pmid = 36158963 | pmc = 9489844 | doi = 10.3389/fneur.2022.922199 | doi-access = free }}</ref> and infectious or post-infectious [[Post viral cerebellar ataxia|cerebellitis]].<ref name="Parvez_2022">{{cite journal | vauthors = Parvez MS, Ohtsuki G | title = Acute Cerebellar Inflammation and Related Ataxia: Mechanisms and Pathophysiology | journal = Brain Sciences | volume = 12 | issue = 3 | page = 367 | date = March 2022 | pmid = 35326323 | pmc = 8946185 | doi = 10.3390/brainsci12030367 | doi-access = free }}</ref>